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The complex of phosphatidylinositol 4,5-bisphosphate and calcium ions is not responsible for Ca(2+)-induced loss of phospholipid asymmetry in the human erythrocyte: a study in Scott syndrome, a disorder of calcium- induced phospholipid scrambling
EM Bevers, T Wiedmer, P Comfurius, J Zhao, EF Smeets, RA Schlegel, AJ Schroit, HJ Weiss, P Williamson, RF Zwaal
Blood (1995) 86 (5): 1983–1991.
Complement-induced vesiculation and exposure of membrane prothrombinase sites in platelets of paroxysmal nocturnal hemoglobinuria
Blood (1993) 82 (4): 1192–1196.
Defective Ca(2+)-induced microvesiculation and deficient expression of procoagulant activity in erythrocytes from a patient with a bleeding disorder: a study of the red blood cells of Scott syndrome
Blood (1992) 79 (2): 380–388.
Participation of protein kinases in complement C5b-9-induced shedding of platelet plasma membrane vesicles
Blood (1991) 78 (11): 2880–2886.
Identification of a platelet dense granule membrane protein that is deficient in a patient with the Hermansky-Pudlak syndrome
Blood (1991) 77 (1): 101–112.
The secretory release reaction initiated by complement proteins C5b-9 occurs without platelet aggregation through glycoprotein IIb-IIIa
Blood (1989) 73 (2): 462–467.
Blood (1986) 68 (4): 875–880.
Repolarization of the membrane potential of blood platelets after complement damage: evidence for a Ca++ -dependent exocytotic elimination of C5b-9 pores
Blood (1986) 68 (2): 556–561.